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What if My 7months Old Baby Is Congested and Has a Fever of 103

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Increased body temperature due to an inflammatory response

Medical status

Fever
Other names Pyrexia, febrile response, febrile[1]
Clinical thermometer 38.7.JPG
An analog medical thermometer showing a temperature of 38.7 °C or 101.7 °F
Specialty Infectious disease, pediatrics
Symptoms Initially: shivering, feeling cold, chills[2]
Afterwards: flushed, sweating[iii]
Complications Delirious seizure[four]
Causes Virus, bacteria, increase in the trunk's temperature set up point[5] [6]
Diagnostic method Temperature > between 37.2 and 38.3 °C (99.0 and 100.9 °F)[one] [7] [viii]
Differential diagnosis Hyperthermia[1]
Treatment Based on underlying cause, not required for fever itself[2] [9]
Medication Ibuprofen, paracetamol (acetaminophen)[ix] [10]
Frequency Common[2] [11]

Fever, also referred to every bit pyrexia, is defined as having a temperature in a higher place the normal range due to an increase in the body'due south temperature set signal.[v] [6] [7] There is not a single agreed-upon upper limit for normal temperature with sources using values between 37.2 and 38.3 °C (99.0 and 100.ix °F) in humans.[ane] [7] [8] The increase in set betoken triggers increased muscle contractions and causes a feeling of cold or chills.[2] This results in greater heat production and efforts to conserve rut.[3] When the set point temperature returns to normal, a person feels hot, becomes flushed, and may begin to sweat.[iii] Rarely a fever may trigger a delirious seizure, with this existence more mutual in immature children.[4] Fevers do non typically become higher than 41 to 42 °C (105.8 to 107.six °F).[6]

A fever can be caused by many medical weather ranging from not-serious to life-threatening.[12] This includes viral, bacterial, and parasitic infections—such as flu, the cold, meningitis, urinary tract infections, appendicitis, COVID-19, and malaria.[12] [13] Non-infectious causes include vasculitis, deep vein thrombosis, connective tissue disease, side furnishings of medication or vaccination, and cancer.[12] [14] It differs from hyperthermia, in that hyperthermia is an increase in body temperature over the temperature set signal, due to either too much heat production or not enough heat loss.[1]

Handling to reduce fever is generally non required.[2] [9] Treatment of associated pain and inflammation, however, may exist useful and help a person rest.[ix] Medications such as ibuprofen or paracetamol (acetaminophen) may help with this as well as lower temperature.[9] [10] Children younger than three months require medical attending, as might people with serious medical problems such as a compromised immune organization or people with other symptoms.[15] Hyperthermia does require treatment.[2]

Fever is one of the about common medical signs.[ii] It is part of about 30% of healthcare visits by children[2] and occurs in upward to 75% of adults who are seriously sick.[eleven] While fever evolved as a defense machinery, treating fever does not appear to worsen outcomes.[16] [17] Fever is often viewed with greater concern by parents and healthcare professionals than is commonly deserved, a phenomenon known every bit fever phobia.[ii] [18]

Associated symptoms [edit]

A fever is ordinarily accompanied by sickness behavior, which consists of lethargy, depression, loss of appetite, sleepiness, hyperalgesia, and the inability to concentrate. Sleeping with a fever can ofttimes crusade intense or confusing nightmares, commonly called "fever dreams".[19] Balmy to severe delirium (which can too cause hallucinations) may too present itself during high fevers.[20]

Diagnosis [edit]

A range for normal temperatures has been found.[8] Cardinal temperatures, such as rectal temperatures, are more accurate than peripheral temperatures.[26] Fever is generally agreed to exist present if the elevated temperature is acquired by a raised set signal and:

  • Temperature in the anus (rectum/rectal) is at or over 37.5–38.3 °C (99.5–100.ix °F)[i] [8] An ear (tympanic) or forehead (temporal) temperature may also exist used.[27] [28]
  • Temperature in the mouth (oral) is at or over 37.2 °C (99.0 °F) in the morn or over 37.7 °C (99.nine °F) in the afternoon[vii] [29]
  • Temperature nether the arm (axillary) is commonly under 0.6 °C (1.1 °F) of the cadre body temperature.[30]

In adults, the normal range of oral temperatures in healthy individuals is 35.vii–37.7 °C (96.3–99.9 °F) among men and 33.ii–38.1 °C (91.viii–100.half-dozen °F) amidst women, while when taken rectally it is 36.7–37.5 °C (98.1–99.v °F) amid men and 36.eight–37.1 °C (98.ii–98.8 °F) among women, and for ear measurement it is 35.v–37.v °C (95.9–99.5 °F) amidst men and 35.7–37.five °C (96.3–99.5 °F) among women.[31]

Normal body temperatures vary depending on many factors, including age, sex activity, time of day, ambience temperature, action level, and more.[32] [33] Normal daily temperature variation has been described equally 0.5 °C (0.9 °F).[seven] : 4012 A raised temperature is non always a fever.[32] For case, the temperature rises in healthy people when they practise, but this is not considered a fever, as the set betoken is normal.[32] On the other paw, a "normal" temperature may be a fever, if information technology is unusually loftier for that person; for example, medically fragile elderly people accept a decreased power to generate body heat, so a "normal" temperature of 37.3 °C (99.1 °F) may represent a clinically significant fever.[32]

Hyperthermia [edit]

Hyperthermia is an tiptop of body temperature over the temperature set point, due to either too much heat production or not enough heat loss.[1] [seven] Hyperthermia is thus not considered fever.[7] : 103 Hyperthermia is not to be confused with hyperpyrexia (which is a very high fever).[vii] : 102

Clinically, information technology is important to distinguish betwixt fever and hyperthermia as hyperthermia may quickly pb to death and does non respond to antipyretic medications. The stardom may notwithstanding be difficult to make in an emergency setting, and is often established by identifying possible causes.[seven] : 103

Types [edit]

Different fever patterns observed in Plasmodium infections.

Diverse patterns of measured patient temperatures have been observed, some of which may be indicative of a particular medical diagnosis:

  • Continuous fever, where temperature remains higher up normal throughout the day and does not fluctuate more than than 1 °C in 24 hours[34] (due east.1000. in bacterial pneumonia, typhoid, infective endocarditis, tuberculosis, or typhus[35]);[36]
  • Intermittent fever, where the temperature elevation is present just for a certain menses, later cycling dorsum to normal (eastward.m., in malaria, leishmaniasis, pyemia, sepsis,[37] or African trypanosomiasis[38]);
  • Remittent fever, where the temperature remains higher up normal throughout the day and fluctuates more than 1 °C in 24 hours[39] (e.g., in infective endocarditis, or brucellosis[twoscore])
  • Pel–Ebstein fever is a cyclic fever that is rarely seen in patients with Hodgkin'south lymphoma.
  • Undulant fever, seen in brucellosis
  • Typhoid fever is an instance of continuous fever and it shows a characteristic footstep-ladder pattern, a step-wise increase in temperature with a loftier plateau.[41]

Among the types of intermittent fever are ones specific to cases of malaria acquired by different pathogens. These are:[42] [43]

  • Quotidian fever, with a 24-hour periodicity, typical of malaria caused past Plasmodium knowlesi (P. knowlesi);[44] [45]
  • Tertian fever, with a 48-hour periodicity, typical of after course malaria caused by P. falciparum, P. vivax, or P. ovale;[42]
  • Quartan fever, with a 72-hour periodicity, typical of later course malaria caused by P. malariae.[42]

In addition, at that place is disagreement regarding whether a specific fever pattern is associated with Hodgkin's lymphoma—the Pel–Ebstein fever, with patients argued to present loftier temperature for one week, followed by depression for the next week, and and so on, where the generality of this pattern is debated.[46] [ needs update ]

Persistent fever that cannot exist explained afterwards repeated routine clinical inquiries is chosen fever of unknown origin.[vii] A neutropenic fever, also called delirious neutropenia, is a fever in the absence of normal allowed system function.[ citation needed ] Considering of the lack of infection-fighting neutrophils, a bacterial infection can spread rapidly; this fever is, therefore, usually considered to crave urgent medical attending.[47] This kind of fever is more commonly seen in people receiving immune-suppressing chemotherapy than in apparently good for you people.[ citation needed ]

Hyperpyrexia [edit]

Hyperpyrexia is an extreme superlative of body temperature which, depending upon the source, is classified as a cadre torso temperature greater than or equal to 40.0 or 41.0 °C (104.0 or 105.8 °F); the range of hyperpyrexias include cases considered severe (≥ xl °C) and extreme (≥ 42 °C).[7] [48] [49] Information technology differs from hyperthermia in that one's thermoregulatory organisation's set point for trunk temperature is set above normal, so heat is generated to achieve it. In contrast, hyperthermia involves torso temperature rising above its ready point due to exterior factors.[7] [50] The high temperatures of hyperpyrexia are considered medical emergencies, as they may indicate a serious underlying condition or atomic number 82 to astringent morbidity (including permanent encephalon damage), or to death.[51] A common crusade of hyperpyrexia is an intracranial hemorrhage.[vii] Other causes in emergency room settings include sepsis, Kawasaki syndrome,[52] neuroleptic malignant syndrome, drug overdose, serotonin syndrome, and thyroid storm.[51]

Differential diagnosis [edit]

Fever is a mutual symptom of many medical atmospheric condition:

  • Infectious disease, eastward.g., COVID-nineteen,[thirteen] Dengue, Ebola, gastroenteritis, HIV, influenza, Lyme disease, malaria, mononucleosis, as well equally infections of the pare, due east.g., abscesses and boils.[53] [54] [55] [56] [57] [58]
  • Immunological diseases, e.1000., relapsing polychondritis,[59] autoimmune hepatitis, granulomatosis with polyangiitis, Horton affliction, inflammatory bowel diseases, Kawasaki disease, lupus erythematosus, sarcoidosis, and Nonetheless's disease;[ citation needed ]
  • Tissue destruction, as a upshot of cerebral bleeding, vanquish syndrome, hemolysis, infarction, rhabdomyolysis, surgery, etc.;[ citation needed ]
  • Cancers, particularly blood cancers such as leukemia and lymphomas;[60]
  • Metabolic disorders, e.g., gout, and porphyria;[ citation needed ] and
  • Inherited metabolic disorder, e.1000., Fabry disease.[7]

Adult and pediatric manifestations for the same illness may differ; for instance, in COVID-19, 1 metastudy describes 92.viii% of adults versus 43.9% of children presenting with fever.[xiii]

In addition, fever tin result from a reaction to an incompatible claret product.[61]

Teething is not a cause of fever.[62]

Selective advantage [edit]

Hyperthermia: Characterized on the left. Normal body temperature (thermoregulatory set point) is shown in green, while the hyperthermic temperature is shown in red. As tin can exist seen, hyperthermia can be conceptualized as an increase above the thermoregulatory fix indicate.
Hypothermia: Characterized in the center: Normal torso temperature is shown in green, while the hypothermic temperature is shown in blue. Equally can be seen, hypothermia can be conceptualized as a decrease below the thermoregulatory fix point.
Fever: Characterized on the right: Normal body temperature is shown in dark-green. It reads "New Normal" considering the thermoregulatory set betoken has risen. This has caused what was the normal body temperature (in blueish) to be considered hypothermic.

Scholars viewing fever from an organismal and evolutionary perspective note the value to an organism of having a fever response, in detail in response to infective disease.[16] [63] [64] On the other mitt, while fever evolved as a defence force mechanism, studies take not been consistent on whether treating fever mostly worsens or improves bloodshed risk in a modern clinical setting.[16] [17] [65] [66] [67] Benefits or harms may depend on the type of infection, wellness condition of the patient & other factors.[16] [65] [66] [67] Studies using warm-blooded vertebrates suggest that they recover more than rapidly from infections or critical disease due to fever.[68] Other studies advise reduced mortality in bacterial infections when fever was present.[69] Fever is thought to contribute to host defense,[16] as the reproduction of pathogens with strict temperature requirements can be hindered, and the rates of some important immunological reactions[ clarification needed ] are increased by temperature.[lxx] Fever has been described in teaching texts as assisting the healing process in various ways, including:

  • increased mobility of leukocytes;[71] : 1044 [ verification needed ]
  • enhanced leukocyte phagocytosis;[71] : 1044 [ verification needed ]
  • decreased endotoxin effects;[71] : 1044 [ verification needed ] and
  • increased proliferation of T cells.[71] : 1044 [ verification needed ] [72] : 212 [ verification needed ]

Pathophysiology [edit]

Hypothalamus [edit]

Temperature is regulated in the hypothalamus. The trigger of a fever, chosen a pyrogen, results in the release of prostaglandin E2 (PGE2). PGE2 in plough acts on the hypothalamus, which creates a systemic response in the trunk, causing heat-generating effects to match a new higher temperature set point. In that location are four receptors in which PGE2 can bind (EP1-iv), with a previous study showing the EP3 subtype is what mediates the fever response.[73] Hence, the hypothalamus can be seen as working like a thermostat.[seven] When the prepare point is raised, the torso increases its temperature through both active generation of heat and retentiveness of heat. Peripheral vasoconstriction both reduces heat loss through the skin and causes the person to feel cold. Norepinephrine increases thermogenesis in brown adipose tissue, and musculus contraction through shivering raises the metabolic rate.[74]

If these measures are insufficient to make the blood temperature in the brain match the new prepare signal in the hypothalamus, the brain orchestrates heat effector mechanisms via the autonomic nervous organization or primary motor heart for shivering. These may be:[ commendation needed ]

  • Increased heat production past increased musculus tone, shivering (muscle movements to produce heat) and release of hormones like epinephrine; and
  • Prevention of heat loss, e.g., through vasoconstriction.

When the hypothalamic set point moves back to baseline—either spontaneously or via medication—normal functions such as sweating, and the reverse of the foregoing processes (eastward.1000., vasodilation, end of shivering, and nonshivering heat product) are used to cool the trunk to the new, lower setting.[ citation needed ]

This contrasts with hyperthermia, in which the normal setting remains, and the torso overheats through undesirable retention of excess heat or over-production of estrus. Hyperthermia is ordinarily the result of an excessively hot environment (heat stroke) or an adverse reaction to drugs. Fever tin exist differentiated from hyperthermia by the circumstances surrounding it and its response to anti-pyretic medications.[vii] [ verification needed ]

In infants, the autonomic nervous system may also activate brown adipose tissue to produce oestrus (non-exercise-associated thermogenesis, also known as not-shivering thermogenesis).[ citation needed ]

Increased middle rate and vasoconstriction contribute to increased claret pressure in fever.[ commendation needed ]

Pyrogens [edit]

A pyrogen is a substance that induces fever.[75] In the presence of an infectious agent, such as leaner, viruses, viroids, etc., the allowed response of the torso is to inhibit their growth and eliminate them. The about common pyrogens are endotoxins, which are lipopolysaccharides (LPS) produced by Gram-negative bacteria such equally E. coli. But pyrogens include non-endotoxic substances (derived from microorganisms other than gram-negative-bacteria or from chemic substances) as well.[76] The types of pyrogens include internal (endogenous) and external (exogenous) to the torso.[ citation needed ]

The "pyrogenicity" of given pyrogens varies: in extreme cases, bacterial pyrogens tin act as superantigens and crusade rapid and dangerous fevers.[77]

Endogenous [edit]

Endogenous pyrogens are cytokines released from monocytes (which are part of the allowed system).[78] In general, they stimulate chemical responses, often in the presence of an antigen, leading to a fever. Whilst they can be a production of external factors like exogenous pyrogens, they can also be induced past internal factors similar damage associated from molecular patterns such equally cases like rheumatoid arthritis or lupus.[79]

Major endogenous pyrogens are interleukin 1 (α and β)[eighty] : 1237–1248 and interleukin 6 (IL-half-dozen).[81] Small-scale endogenous pyrogens include interleukin-8, tumor necrosis factor-β, macrophage inflammatory poly peptide-α and macrophage inflammatory protein-β too as interferon-α, interferon-β, and interferon-γ.[80] : 1237–1248 Tumor necrosis factor-α (TNF) likewise acts as a pyrogen, mediated by interleukin 1 (IL-1) release.[82] These cytokine factors are released into full general circulation, where they migrate to the brain'due south circumventricular organs where they are more than easily absorbed than in areas protected by the blood–brain barrier.[ citation needed ] The cytokines so bind to endothelial receptors on vessel walls to receptors on microglial cells, resulting in activation of the arachidonic acid pathway.[ citation needed ]

Of these, IL-1β, TNF, and IL-6 are able to raise the temperature setpoint of an organism and cause fever. These proteins produce a cyclooxygenase which induces the hypothalamic production of PGE2 which then stimulates the release of neurotransmitters such as circadian adenosine monophosphate and increases body temperature.[83]

Exogenous [edit]

Exogenous pyrogens are external to the body and are of microbial origin. In general, these pyrogens, including bacterial cell wall products, may human activity on Toll-similar receptors in the hypothalamus and drag the thermoregulatory setpoint.[84]

An example of a class of exogenous pyrogens are bacterial lipopolysaccharides (LPS) nowadays in the cell wall of gram-negative bacteria. According to one mechanism of pyrogen activeness, an immune organization protein, lipopolysaccharide-binding protein (LBP), binds to LPS, and the LBP–LPS complex then binds to a CD14 receptor on a macrophage. The LBP-LPS binding to CD14 results in cellular synthesis and release of various endogenous cytokines, due east.g., interleukin 1 (IL-1), interleukin half-dozen (IL-vi), and tumor necrosis factor-alpha (TNFα). A further downstream event is activation of the arachidonic acid pathway.[85]

PGE2 release [edit]

PGE2 release comes from the arachidonic acid pathway. This pathway (as information technology relates to fever), is mediated by the enzymes phospholipase A2 (PLA2), cyclooxygenase-2 (COX-2), and prostaglandin E2 synthase. These enzymes ultimately mediate the synthesis and release of PGE2.[ citation needed ]

PGE2 is the ultimate mediator of the delirious response. The setpoint temperature of the torso will remain elevated until PGE2 is no longer nowadays. PGE2 acts on neurons in the preoptic area (POA) through the prostaglandin E receptor three (EP3). EP3-expressing neurons in the POA innervate the dorsomedial hypothalamus (DMH), the rostral raphe pallidus nucleus in the medulla oblongata (rRPa), and the paraventricular nucleus (PVN) of the hypothalamus. Fever signals sent to the DMH and rRPa lead to stimulation of the sympathetic output organisation, which evokes non-shivering thermogenesis to produce torso heat and peel vasoconstriction to subtract rut loss from the trunk surface. It is presumed that the innervation from the POA to the PVN mediates the neuroendocrine furnishings of fever through the pathway involving pituitary gland and various endocrine organs.[ citation needed ]

Management [edit]

Fever does non necessarily need to be treated,[86] and most people with a fever recover without specific medical attention.[87] Although it is unpleasant, fever rarely rises to a dangerous level fifty-fifty if untreated. Damage to the encephalon generally does not occur until temperatures reach 42 °C (107.6 °F), and it is rare for an untreated fever to exceed twoscore.6 °C (105 °F).[88] Treating fever in people with sepsis does not affect outcomes.[89]

Conservative measures [edit]

Limited bear witness supports sponging or bathing feverish children with tepid water.[90] The utilise of a fan or air conditioning may somewhat reduce the temperature and increase comfort. If the temperature reaches the extremely loftier level of hyperpyrexia, aggressive cooling is required (more often than not produced mechanically via conduction past applying numerous ice packs across most of the body or straight submersion in ice water).[51] In general, people are brash to keep adequately hydrated.[91] Whether increased fluid intake improves symptoms or shortens respiratory illnesses such every bit the common cold is non known.[92]

Medications [edit]

Medications that lower fevers are called antipyretics. The antipyretic ibuprofen is constructive in reducing fevers in children.[93] It is more effective than acetaminophen (paracetamol) in children.[93] Ibuprofen and acetaminophen may be safely used together in children with fevers.[94] [95] The efficacy of acetaminophen by itself in children with fevers has been questioned.[96] Ibuprofen is also superior to aspirin in children with fevers.[97] Additionally, aspirin is not recommended in children and young adults (those under the age of 16 or 19 depending on the country) due to the risk of Reye'southward syndrome.[98]

Using both paracetamol and ibuprofen at the same time or alternating betwixt the two is more effective at decreasing fever than using only paracetamol or ibuprofen.[99] Information technology is not clear if it increases child comfort.[99] Response or nonresponse to medications does non predict whether or non a child has a serious illness.[100]

With respect to the effect of antipyretics on the gamble of death in those with infection, studies accept found mixed results as of 2019.[101] Animal models accept found worsened outcomes with the use of antipyretics in influenza as of 2010 merely they have non been studied for this use in humans.[102]

Epidemiology [edit]

Fever is one of the most mutual medical signs.[2] It is role of near 30% of healthcare visits by children,[two] and occurs in upward to 75% of adults who are seriously sick.[eleven] Almost v% of people who go to an emergency room have a fever.[103]

History [edit]

A number of types of fever were known as early as 460 BC to 370 BC when Hippocrates was practicing medicine including that due to malaria (tertian or every two days and quartan or every three days).[104] Information technology also became clear effectually this time that fever was a symptom of disease rather than a disease in and of itself.[104]

Infections presenting with fever were a major source of bloodshed in humans for about 200,000 years. Until the belatedly nineteenth century, approximately half of all humans died from infections earlier the age of xv.[105]

An older term, febricula (a atomic form of the Latin give-and-take for fever), was once used to refer to a depression-grade fever lasting but a few days. This term fell out of use in the early 20th century, and the symptoms it referred to are now thought to have been caused mainly past various small-scale viral respiratory infections.[106]

Society and civilization [edit]

Fever is often viewed with greater business organization by parents and healthcare professionals than might be deserved, a phenomenon known every bit fever phobia,[2] [107] which is based in both caregiver's and parents' misconceptions well-nigh fever in children. Among them, many parents incorrectly believe that fever is a affliction rather than a medical sign, that fifty-fifty low fevers are harmful, and that any temperature even briefly or slightly higher up the oversimplified "normal" number marked on a thermometer is a clinically significant fever.[107] They are also agape of harmless side furnishings like febrile seizures and dramatically overestimate the likelihood of permanent damage from typical fevers.[107] The underlying trouble, according to professor of pediatrics Barton D. Schmitt, is that "as parents nosotros tend to suspect that our children's brains may melt."[108] Equally a result of these misconceptions parents are anxious, give the child fever-reducing medicine when the temperature is technically normal or merely slightly elevated, and interfere with the child's sleep to give the child more medicine.[107]

Other species [edit]

Fever is an important characteristic for the diagnosis of affliction in domestic animals. The torso temperature of animals, which is taken rectally, is different from one species to another. For example, a horse is said to have a fever above 101 °F ( 38.3 °C).[109] In species that allow the trunk to have a broad range of "normal" temperatures, such every bit camels,[110] whose torso temperature varies as the environmental temperature varies,[111] the torso temperature which constitutes a delirious state differs depending on the ecology temperature.[112] Fever can also exist behaviorally induced by invertebrates that do not have immune-system based fever. For instance, some species of grasshopper volition thermoregulate to reach body temperatures that are 2–5 °C higher than normal in gild to inhibit the growth of fungal pathogens such as Beauveria bassiana and Metarhizium acridum.[113] Honeybee colonies are likewise able to induce a fever in response to a fungal parasite Ascosphaera apis.[113]

References [edit]

  1. ^ a b c d east f g h i Axelrod YK, Diringer MN (May 2008). "Temperature management in acute neurologic disorders". Neurologic Clinics. 26 (2): 585–603, eleven. doi:10.1016/j.ncl.2008.02.005. PMID 18514828.
  2. ^ a b c d e f 1000 h i j chiliad l Sullivan JE, Farrar HC (March 2011). "Fever and antipyretic use in children". Pediatrics. 127 (three): 580–87. doi:10.1542/peds.2010-3852. PMID 21357332.
  3. ^ a b c Huether, Sue E. (2014). Pathophysiology: The Biologic Footing for Disease in Adults and Children (7th ed.). Elsevier Health Sciences. p. 498. ISBN978-0323293754.
  4. ^ a b CDC Staff (31 March 2020). "Taking Intendance of Someone Who is Sick: Caring for Someone Sick at Abode". Archived from the original on 24 March 2015. Retrieved eight May 2015.
  5. ^ a b Kluger MJ (2015). Fever: Its Biological science, Evolution, and Function. Princeton University Press. p. 57. ISBN978-1400869831.
  6. ^ a b c Garmel GM, Mahadevan SV, eds. (2012). "Fever in adults". An introduction to clinical emergency medicine (second ed.). Cambridge: Cambridge Academy Printing. p. 375. ISBN978-0521747769.
  7. ^ a b c d e f yard h i j thou l chiliad northward o p Dinarello CA, Porat R (2018). "Chapter xv: Fever". In Jameson JL, Fauci AS, Kasper DL, Hauser SL, Longo DL, Loscalzo, J (eds.). Harrison's Principles of Internal Medicine. Vol. i–2 (20th ed.). New York, NY: McGraw-Hill. ISBN9781259644030 . Retrieved 31 March 2020.
  8. ^ a b c d e f Laupland KB (July 2009). "Fever in the critically sick medical patient". Critical Care Medicine. 37 (7 Suppl): S273-viii. doi:ten.1097/CCM.0b013e3181aa6117. PMID 19535958.
  9. ^ a b c d e
  10. ^ a b Garmel GM, Mahadevan SV, eds. (2012). An introduction to clinical emergency medicine (2nd ed.). Cambridge: Cambridge University Printing. p. 401. ISBN978-0521747769.
  11. ^ a b c Kiekkas P, Aretha D, Bakalis Northward, Karpouhtsi I, Marneras C, Baltopoulos GI (Baronial 2013). "Fever effects and handling in critical intendance: literature review". Australian Critical Intendance. 26 (3): 130–35. doi:10.1016/j.aucc.2012.x.004. PMID 23199670.
  12. ^ a b c Garmel GM, Mahadevan SV, eds. (2012). An introduction to clinical emergency medicine (second ed.). Cambridge: Cambridge University Press. p. five. ISBN978-0521747769.
  13. ^ a b c Rodriguez-Morales AJ, Cardona-Ospina JA, Gutiérrez-Ocampo E, Villamizar-Peña R, Holguin-Rivera Y, Escalera-Antezana JP, Alvarado-Arnez LE, Bonilla-Aldana DK, Franco-Paredes C (13 March 2020). "Clinical, laboratory and imaging features of COVID-19: A systematic review and meta-analysis". Travel Medicine and Infectious Affliction. 34: 101623. doi:x.1016/j.tmaid.2020.101623. PMC7102608. PMID 32179124.
  14. ^ Dayal R, Agarwal D (Jan 2016). "Fever in Children and Fever of Unknown Origin". Indian Journal of Pediatrics. 83 (1): 38–43. doi:10.1007/s12098-015-1724-iv. PMID 25724501. S2CID 34481402.
  15. ^ "Fever". MedlinePlus. thirty August 2014. Archived from the original on 11 May 2009.
  16. ^ a b c d due east Schaffner A (March 2006). "Fieber – nützliches oder schädliches, zu behandelndes Symptom?" [Fever–useful or noxious symptom that should be treated?]. Therapeutische Umschau (in German). 63 (3): 185–88. doi:10.1024/0040-5930.63.3.185. PMID 16613288. Abstract alone is in German language and in English.
  17. ^ a b Niven DJ, Stelfox HT, Laupland KB (June 2013). "Antipyretic therapy in delirious critically ill adults: A systematic review and meta-analysis". Journal of Critical Care. 28 (three): 303–10. doi:10.1016/j.jcrc.2012.09.009. PMID 23159136.
  18. ^ Crocetti Grand, Moghbeli North, Serwint J (June 2001). "Fever Phobia Revisited: Accept Parental Misconceptions About Fever Inverse in 20 Years?". Pediatrics. 107 (six): 1241–1246. doi:10.1542/peds.107.6.1241. PMID 11389237.
  19. ^ Kelley KW, Bluthé RM, Dantzer R, Zhou JH, Shen WH, Johnson RW, Broussard SR (Feb 2003). "Cytokine-induced sickness behavior". Brain, Behavior, and Immunity. 17 Suppl 1 (1): S112–eighteen. doi:10.1016/S0889-1591(02)00077-vi. PMID 12615196. S2CID 25400611.
  20. ^ Adamis D, Treloar A, Martin FC, Macdonald AJ (December 2007). "A brief review of the history of delirium as a mental disorder". History of Psychiatry. 18 (72 Pt 4): 459–69. doi:10.1177/0957154X07076467. PMID 18590023. S2CID 24424207.
  21. ^ Marx J (2006). Rosen'southward emergency medicine : concepts and clinical practice (6th ed.). Philadelphia: Mosby/Elsevier. p. 2239. ISBN978-0-323-02845-5. OCLC 58533794.
  22. ^ Hutchison JS, Ward RE, Lacroix J, Hébert PC, Barnes MA, Bohn DJ, et al. (June 2008). "Hypothermia therapy after traumatic brain injury in children". The New England Journal of Medicine. 358 (23): 2447–56. doi:x.1056/NEJMoa0706930. PMID 18525042.
  23. ^ Pryor JA, Prasad Equally (2008). Physiotherapy for Respiratory and Cardiac Problems: Adults and Paediatrics. Elsevier Health Sciences. p. eight. ISBN978-0702039744. Torso temperature is maintained inside the range 36.5-37.5 °C. It is lowest in the early morning and highest in the afternoon.
  24. ^ Grunau BE, Wiens MO, Brubacher JR (September 2010). "Dantrolene in the handling of MDMA-related hyperpyrexia: a systematic review". Cjem. 12 (v): 435–42. doi:10.1017/s1481803500012598. PMID 20880437. Dantrolene may also exist associated with improved survival and reduced complications, specially in patients with farthermost (≥ 42 °C) or astringent (≥ xl °C) hyperpyrexia
  25. ^ Sharma HS, ed. (2007). Neurobiology of Hyperthermia (1st ed.). Elsevier. pp. 175–177, 485. ISBN9780080549996 . Retrieved xix Nov 2016. Despite the myriad of complications associated with estrus affliction, an height of cadre temperature in a higher place 41.0 °C (oft referred to as fever or hyperpyrexia) is the most widely recognized symptom of this syndrome.
  26. ^ Niven DJ, Gaudet JE, Laupland KB, Mrklas KJ, Roberts DJ, Stelfox HT (November 2015). "Accuracy of peripheral thermometers for estimating temperature: a systematic review and meta-analysis". Register of Internal Medicine. 163 (10): 768–77. doi:10.7326/M15-1150. PMID 26571241. S2CID 4004360.
  27. ^ "Measuring a Baby'south Temperature". world wide web.hopkinsmedicine.org. Archived from the original on 3 Nov 2019. Retrieved 10 September 2019.
  28. ^ "Tips for taking your child's temperature". Mayo Clinic . Retrieved ten September 2019.
  29. ^ Barone JE (August 2009). "Fever: Fact and fiction". The Journal of Trauma. 67 (2): 406–09. doi:10.1097/TA.0b013e3181a5f335. PMID 19667898.
  30. ^ Pecoraro, Valentina; Petri, Davide; Costantino, Giorgio; Squizzato, Alessandro; Moja, Lorenzo; Virgili, Gianni; Lucenteforte, Ersilia (25 November 2020). "The diagnostic accuracy of digital, infrared and mercury-in-glass thermometers in measuring torso temperature: a systematic review and network meta-analysis". Internal and Emergency Medicine. Springer Scientific discipline and Business Media LLC. 16 (4): 1071–1083. doi:ten.1007/s11739-020-02556-0. ISSN 1828-0447. PMC7686821. PMID 33237494.
  31. ^ Sund-Levander G, Forsberg C, Wahren LK (June 2002). "Normal oral, rectal, tympanic and axillary body temperature in adult men and women: a systematic literature review". Scandinavian Journal of Caring Sciences. xvi (2): 122–28. doi:10.1046/j.1471-6712.2002.00069.10. PMID 12000664.
  32. ^ a b c d Garami, András; Székely, Miklós (vi May 2014). "Body temperature". Temperature: Multidisciplinary Biomedical Journal. one (1): 28–29. doi:10.4161/temp.29060. ISSN 2332-8940. PMC4972507. PMID 27583277.
  33. ^ "Body temperature: What is the new normal?". www.medicalnewstoday.com. 12 January 2020. Retrieved 7 Apr 2020.
  34. ^ Ogoina D (Baronial 2011). "Fever, fever patterns and diseases chosen 'fever' – a review". Periodical of Infection and Public Health. four (iii): 108–24. doi:10.1016/j.jiph.2011.05.002. PMID 21843857.
  35. ^ Typhoid fever may show a specific fever pattern, a Wunderlich curve, with a slow stepwise increase and a high plateau (drops due to fever-reducing drugs are excluded).[ citation needed ]
  36. ^ Dall, Lawrence; Stanford, James F. (1990). "Fever, Chills, and Dark Sweats". In Walker, H. Kenneth; Hall, W. Dallas; Hurst, J. Willis (eds.). Clinical Methods: The History, Physical, and Laboratory Examinations (3rd ed.). Boston: Butterworths. ISBN0-409-90077-X. PMID 21250166.
  37. ^ Inayatullah, Muhammad; Nasir, Shabbir Ahmed (2016). Bedside Techniques: Methods of Clinical Test (fourth ed.). ISBN978-969-494-920-8. [ page needed ]
  38. ^ "CDC - African Trypanosomiasis - Illness". www.cdc.gov. 28 Apr 2020. Retrieved xviii July 2021.
  39. ^ The Medical News and Library. Lea & Blanchard. 1843. p. 96 – via Internet Annal. remittent fever news.
  40. ^ "Brucella/Brucellosis". The Lecturio Medical Concept Library . Retrieved 19 July 2021.
  41. ^ "Enteric Fever (Typhoid Fever)". The Lecturio Medical Concept Library. 27 August 2020. Retrieved 19 July 2021.
  42. ^ a b c Ferri FF (2009). "Affiliate 332. Protozoal infections". Ferri'due south Color Atlas and Text of Clinical Medicine. Elsevier Health Sciences. pp. 1159ff. ISBN9781416049197. Archived from the original on 3 June 2016. Retrieved 31 March 2020.
  43. ^ Muhammad I, Nasir, SA (2009). Bedside Techniques: Methods of Clinical Examination. Multan, Pakistan: Saira Publishers/Salamat Iqbal Printing. [ page needed ] [ amend source needed ]
  44. ^ Singh, B.; Daneshvar, C. (one Apr 2013). "Man Infections and Detection of Plasmodium knowlesi". Clinical Microbiology Reviews. 26 (two): 165–184. doi:10.1128/CMR.00079-12. PMC3623376. PMID 23554413.
  45. ^ Chin, Due west.; Contacos, P. Thou.; Coatney, K. R.; Kimball, H. R. (20 August 1965). "A Naturally Acquired Quotidian-Type Malaria in Human being Transferable to Monkeys". Science. 149 (3686): 865. Bibcode:1965Sci...149..865C. doi:10.1126/science.149.3686.865. PMID 14332847. S2CID 27841173.
  46. ^ Hilson AJ (July 1995). "Pel-Ebstein fever". The New England Periodical of Medicine. 333 (ane): 66–67. doi:10.1056/NEJM199507063330118. PMID 7777006. , which cites Richard Asher'south lecture, "Making Sense" [Lancet (1959) 2: 359].
  47. ^ White, Lindsey; Ybarra, Michael (1 December 2017). "Neutropenic Fever". Hematology/Oncology Clinics of North America. 31 (6): 981–993. doi:10.1016/j.hoc.2017.08.004. PMID 29078933 – via ClinicalKey.
  48. ^ Grunau BE, Wiens MO, Brubacher JR (September 2010). "Dantrolene in the treatment of MDMA-related hyperpyrexia: a systematic review". Canadian Journal of Emergency Medicine. 12 (v): 435–42. doi:10.1017/s1481803500012598. PMID 20880437. Dantrolene may also be associated with improved survival and reduced complications, especially in patients with extreme (≥ 42 °C) or astringent (≥ 40 °C) hyperpyrexia
  49. ^ Sharma HS, ed. (2007). Neurobiology of Hyperthermia (1st ed.). Elsevier. pp. 175–77, 485. ISBN978-0080549996. Archived from the original on 8 September 2017. Retrieved 19 November 2016. Despite the myriad of complications associated with estrus illness, an elevation of cadre temperature above 41.0 °C (often referred to equally fever or hyperpyrexia) is the most widely recognized symptom of this syndrome.
  50. ^ See section in Chapter 15 therein, the department on "Fever versus hyperthermia".
  51. ^ a b c McGugan EA (March 2001). "Hyperpyrexia in the emergency department". Emergency Medicine. 13 (1): 116–20. doi:10.1046/j.1442-2026.2001.00189.x. PMID 11476402.
  52. ^ Marx (2006), p. 2506.
  53. ^ Raoult, Didier; Levy, Pierre-Yves; Dupont, Hervé Tissot; Chicheportiche, Colette; Tamalet, Catherine; Gastaut, Jean-Albert; Salducci, Jacques (Jan 1993). "Q fever and HIV infection". AIDS. 7 (1): 81–86. doi:ten.1097/00002030-199301000-00012. ISSN 0269-9370. PMID 8442921.
  54. ^ French, Neil; Nakiyingi, Jessica; Lugada, Eric; Watera, Christine; Whitworth, James A. G.; Gilks, Charles F. (May 2001). "Increasing rates of malarial fever with deteriorating immune status in HIV-ane-infected Ugandan adults". AIDS. 15 (7): 899–906. doi:x.1097/00002030-200105040-00010. ISSN 0269-9370. PMID 11399962. S2CID 25470703. Archived from the original on 22 Feb 2022.
  55. ^ Heymann, D. 50.; Weisfeld, J. S.; Webb, P. A.; Johnson, G. M.; Cairns, T.; Berquist, H. (1 September 1980). "Ebola Hemorrhagic Fever: Tandala, Zaire, 1977-1978". Journal of Infectious Diseases. 142 (three): 372–376. doi:10.1093/infdis/142.3.372. ISSN 0022-1899. PMID 7441008.
  56. ^ Feldmann, Heinz; Geisbert, Thomas W (March 2011). "Ebola haemorrhagic fever". The Lancet. 377 (9768): 849–862. doi:10.1016/s0140-6736(ten)60667-8. ISSN 0140-6736. PMC3406178. PMID 21084112.
  57. ^ Oakley, Miranda S.; Gerald, Noel; McCutchan, Thomas F.; Aravind, L.; Kumar, Sanjai (October 2011). "Clinical and molecular aspects of malaria fever". Trends in Parasitology. 27 (ten): 442–449. doi:10.1016/j.pt.2011.06.004. ISSN 1471-4922. PMID 21795115.
  58. ^ Colunga-Salas, Pablo; Sánchez-Montes, Sokani; Volkow, Patricia; Ruíz-Remigio, Adriana; Becker, Ingeborg (17 September 2020). "Lyme disease and relapsing fever in Mexico: An overview of human and wildlife infections". PLOS I. xv (9): e0238496. Bibcode:2020PLoSO..1538496C. doi:ten.1371/periodical.pone.0238496. ISSN 1932-6203. PMC7497999. PMID 32941463.
  59. ^ Puéchal X, Terrier B, Mouthon L, Costedoat-Chalumeau N, Guillevin L, Le Jeunne C (March 2014). "Relapsing polychondritis". Articulation, Bone, Spine. 81 (2): 118–24. doi:10.1016/j.jbspin.2014.01.001. PMID 24556284.
  60. ^ "Signs and Symptoms of Cancer | Do I Accept Cancer?". www.cancer.org . Retrieved xx June 2020.
  61. ^ Dean, Laura (2005). Blood transfusions and the allowed system. National Center for Biotechnology Information (The states).
  62. ^ Massignan C, Cardoso M, Porporatti AL, Aydinoz South, Canto Chiliad, Mezzomo LA, Bolan M (March 2016). "Signs and Symptoms of Master Tooth Eruption: A Meta-assay". Pediatrics. 137 (3): e20153501. doi:10.1542/peds.2015-3501. PMID 26908659. Archived from the original on 21 February 2016.
  63. ^ SoszyÅ„ski D (2003). "[The pathogenesis and the adaptive value of fever]". PostÄ™py Higieny I Medycyny DoÅ›wiadczalnej (in Polish). 57 (5): 531–54. PMID 14737969.
  64. ^ Kluger MJ, Kozak W, Conn CA, Leon LR, Soszynski D (September 1998). "Function of fever in disease". Annals of the New York University of Sciences. 856 (1): 224–33. Bibcode:1998NYASA.856..224K. doi:x.1111/j.1749-6632.1998.tb08329.x. PMID 9917881. S2CID 12408561.
  65. ^ a b Schulman, Carl I.; Namias, Nicholas; Doherty, James; Manning, Ronald J.; Li, Pamela; Elhaddad, Ahmed; Lasko, David; Amortegui, Jose; Dy, Christopher J.; Dlugasch, Lucie; Baracco, Gio; Cohn, Stephen M. (2005). "The Outcome of Antipyretic Therapy upon Outcomes in Critically Sick Patients: A Randomized, Prospective Study". Surgical Infections. 6 (iv): 369–375. doi:10.1089/sur.2005.6.369. PMID 16433601.
  66. ^ a b Drewry, Anne M.; Ablordeppey, Enyo A.; Murray, Ellen T.; Stoll, Carolyn R. T.; Izadi, Sonya R.; Dalton, Catherine M.; Hardi, Angela C.; Fowler, Susan A.; Fuller, Brian M.; Colditz, Graham A. (2017). "Antipyretic Therapy in Critically Ill Septic Patients". Critical Care Medicine. 45 (v): 806–813. doi:10.1097/CCM.0000000000002285. PMC5389594. PMID 28221185.
  67. ^ a b https://www.australiancriticalcare.com/article/S1036-7314(nineteen)30093-ane/fulltext
  68. ^ Su F, Nguyen ND, Wang Z, Cai Y, Rogiers P, Vincent JL (June 2005). "Fever command in septic stupor: beneficial or harmful?". Shock. 23 (6): 516–20. PMID 15897803.
  69. ^ Rantala South, Vuopio-Varkila J, Vuento R, Huhtala H, Syrjänen J (April 2009). "Predictors of mortality in beta-hemolytic streptococcal bacteremia: a population-based report". The Periodical of Infection. 58 (4): 266–72. doi:10.1016/j.jinf.2009.01.015. PMID 19261333.
  70. ^ Fischler MP, Reinhart WH (May 1997). "[Fever: friend or enemy?]". Schweizerische Medizinische Wochenschrift (in German). 127 (twenty): 864–70. PMID 9289813.
  71. ^ a b c d Craven RF, Hirnle CJ (2003). Fundamentals of Nursing: Human Health and Function (4th ed.). Philadelphia, PA: Lippincott Williams & Wilkins. ISBN9780781758185 . Retrieved two April 2020. {{cite book}}: CS1 maint: uses authors parameter (link)
  72. ^ Lewis SM, Dirksen SR, Heitkemper MM (2005). Medical-Surgical Nursing: Cess and Management of Clinical Issues (6th ed.). Amsterdam, NL: Elsevier-Health Sciences. ISBN9780323031059 . Retrieved 2 Apr 2020. {{cite book}}: CS1 maint: uses authors parameter (link)
  73. ^ Ushikubi F, et al. (September 1998). "Impaired febrile response in mice lacking the prostaglandin E receptor subtype EP3". Nature. 395 (6699): 281–284. Bibcode:1998Natur.395..281U. doi:10.1038/26233. PMID 9751056. S2CID 4420632.
  74. ^ Evans SS, Repasky EA, Fisher DT (June 2015). "Fever and the thermal regulation of immunity: the allowed organization feels the heat". Nature Reviews. Immunology. 15 (6): 335–49. doi:x.1038/nri3843. PMC4786079. PMID 25976513.
  75. ^ Hagel L, Jagschies Thou, Sofer Thousand (1 Jan 2008). "5 - Analysis". Handbook of Procedure Chromatography (2nd ed.). Academic Press. pp. 127–145. doi:x.1016/b978-012374023-6.50007-five. ISBN978-0-12-374023-six.
  76. ^ Kojima 1000 (1 January 2012). "17 - Biological evaluation and regulation of medical devices in Nippon". In Boutrand J (ed.). Biocompatibility and Functioning of Medical Devices . Woodhead Publishing Serial in Biomaterials. Woodhead Publishing. pp. 404–448. doi:10.1533/9780857096456.4.404. ISBN978-0-85709-070-6.
  77. ^ Affairs, Office of Regulatory (iii Nov 2018). "Pyrogens, Nonetheless a Danger". FDA.
  78. ^ Constable PD, Hinchcliff KW, Done SH, Grünberg W, eds. (i Jan 2017). "4 - Full general Systemic States". Veterinary Medicine (11th ed.). West.B. Saunders. pp. 43–112. doi:10.1016/b978-0-7020-5246-0.00004-8. ISBN978-0-7020-5246-0. S2CID 214758182.
  79. ^ Dinarello CA (31 March 2015). "The history of fever, leukocytic pyrogen and interleukin-one". Temperature. 2 (one): 8–xvi. doi:x.1080/23328940.2015.1017086. PMC4843879. PMID 27226996.
  80. ^ a b Stitt, John (2008). "Chapter 59: Regulation of Body Temperature". In Boron WF, Boulpaep, EL (eds.). Medical Physiology: A Cellular and Molecular Arroyo (second ed.). Philadelphia, PA: Elsevier Saunders. ISBN9781416031154 . Retrieved 2 April 2020.
  81. ^ Murphy, Kenneth (Kenneth M.) (2017). Janeway's immunobiology. Weaver, Casey (ninth ed.). New York, NY, USA. pp. 118–119. ISBN978-0-8153-4505-three. OCLC 933586700.
  82. ^ Stefferl A, Hopkins SJ, Rothwell NJ, Luheshi GN (Baronial 1996). "The office of TNF-blastoff in fever: opposing actions of homo and murine TNF-alpha and interactions with IL-beta in the rat". British Journal of Pharmacology. 118 (8): 1919–24. doi:10.1111/j.1476-5381.1996.tb15625.x. PMC1909906. PMID 8864524.
  83. ^ Srinivasan L, Harris MC, Kilpatrick LE (1 Jan 2017). "128 - Cytokines and Inflammatory Response in the Fetus and Neonate". In Polin RA, Abman SH, Rowitch DH, Benitz WE (eds.). Fetal and Neonatal Physiology (5th ed.). Elsevier. pp. 1241–1254.e4. doi:x.1016/b978-0-323-35214-7.00128-1. ISBN978-0-323-35214-7.
  84. ^ Wilson ME, Boggild AK (ane Jan 2011). "130 - Fever and Systemic Symptoms". In Guerrant RL, Walker DH, Weller PF (eds.). Tropical Infectious Diseases: Principles, Pathogens and Practice (tertiary ed.). W.B. Saunders. pp. 925–938. doi:10.1016/b978-0-7020-3935-5.00130-0. ISBN978-0-7020-3935-5.
  85. ^ Roth J, Blatteis CM (October 2014). "Mechanisms of fever product and lysis: Lessons from experimental LPS fever". Comprehensive Physiology. 4 (4): 1563–604. doi:10.1002/cphy.c130033. ISBN9780470650714. PMID 25428854.
  86. ^ Ludwig J, McWhinnie H (May 2019). "Antipyretic drugs in patients with fever and infection: literature review". Br J Nurs. 28 (ten): 610–618. doi:ten.12968/bjon.2019.28.10.610. PMID 31116598. S2CID 162182092.
  87. ^ "What To Do If You Become Sick: 2009 H1N1 and Seasonal Influenza". Centers for Disease Control and Prevention. 7 May 2009. Archived from the original on 3 November 2009. Retrieved one November 2009.
  88. ^ Edward James Walter and Mike Carraretto (2016). "The neurological and cognitive consequences of hyperthermia". Critical Care. 20 (1): 199. doi:10.1186/s13054-016-1376-four. PMC4944502. PMID 27411704. {{cite periodical}}: CS1 maint: uses authors parameter (link)
  89. ^ Drewry AM, Ablordeppey EA, Murray ET, Stoll CR, Izadi SR, Dalton CM, Hardi Air conditioning, Fowler SA, Fuller BM, Colditz GA (May 2017). "Antipyretic Therapy in Critically Ill Septic Patients: A Systematic Review and Meta-Analysis". Critical Care Medicine. 45 (five): 806–13. doi:10.1097/CCM.0000000000002285. PMC5389594. PMID 28221185.
  90. ^ Meremikwu One thousand, Oyo-Ita A (2003). Meremikwu MM (ed.). "Physical methods for treating fever in children". The Cochrane Database of Systematic Reviews (2): CD004264. doi:10.1002/14651858.CD004264. PMC6532675. PMID 12804512.
  91. ^ "Fever". National Institute of Wellness. Archived from the original on xxx Apr 2016.
  92. ^ Guppy MP, Mickan SM, Del Mar CB, Thorning Due south, Rack A (February 2011). "Advising patients to increase fluid intake for treating acute respiratory infections". The Cochrane Database of Systematic Reviews (2): CD004419. doi:10.1002/14651858.CD004419.pub3. PMC7197045. PMID 21328268.
  93. ^ a b Perrott DA, Piira T, Goodenough B, Champion GD (June 2004). "Efficacy and prophylactic of acetaminophen vs ibuprofen for treating children's pain or fever: a meta-assay". Archives of Pediatrics & Adolescent Medicine. 158 (half-dozen): 521–26. doi:10.1001/archpedi.158.6.521. PMID 15184213.
  94. ^ Hay AD, Redmond NM, Costelloe C, Montgomery AA, Fletcher M, Hollinghurst South, Peters TJ (May 2009). "Paracetamol and ibuprofen for the handling of fever in children: the PITCH randomised controlled trial". Health Technology Assessment. 13 (27): iii–4, ix–10, one–163. doi:x.3310/hta13270. PMID 19454182.
  95. ^ Southey ER, Soares-Weiser K, Kleijnen J (September 2009). "Systematic review and meta-assay of the clinical rubber and tolerability of ibuprofen compared with paracetamol in paediatric pain and fever". Current Medical Enquiry and Opinion. 25 (9): 2207–22. doi:10.1185/03007990903116255. PMID 19606950. S2CID 31653539.
  96. ^ Meremikwu M, Oyo-Ita A (2002). "Paracetamol for treating fever in children". The Cochrane Database of Systematic Reviews (2): CD003676. doi:10.1002/14651858.CD003676. PMC6532671. PMID 12076499.
  97. ^ Autret E, Reboul-Marty J, Henry-Launois B, Laborde C, Courcier S, Goehrs JM, Languillat G, Launois R (1997). "Evaluation of ibuprofen versus aspirin and paracetamol on efficacy and comfort in children with fever". European Journal of Clinical Pharmacology. 51 (5): 367–71. doi:10.1007/s002280050215. PMID 9049576. S2CID 27519225.
  98. ^ "2.9 Antiplatelet drugs". British National Formulary for Children. British Medical Association and Royal Pharmaceutical Society of Keen Britain. 2007. p. 151.
  99. ^ a b Wong T, Stang AS, Ganshorn H, Hartling L, Maconochie IK, Thomsen AM, Johnson DW (October 2013). "Combined and alternating paracetamol and ibuprofen therapy for delirious children". The Cochrane Database of Systematic Reviews (ten): CD009572. doi:10.1002/14651858.CD009572.pub2. PMC6532735. PMID 24174375.
  100. ^ Male monarch D (August 2013). "Question 2: does a failure to respond to antipyretics predict serious illness in children with a fever?". Archives of Disease in Babyhood. 98 (8): 644–46. doi:10.1136/archdischild-2013-304497. PMID 23846358. S2CID 32438262.
  101. ^ Ludwig J, McWhinnie H (May 2019). "Antipyretic drugs in patients with fever and infection: literature review". British Journal of Nursing. 28 (10): 610–618. doi:10.12968/bjon.2019.28.x.610. PMID 31116598. S2CID 162182092.
  102. ^ Eyers S, Weatherall M, Shirtcliffe P, Perrin K, Beasley R (October 2010). "The upshot on mortality of antipyretics in the handling of influenza infection: systematic review and meta-analysis". Journal of the Royal Society of Medicine. 103 (10): 403–11. doi:x.1258/jrsm.2010.090441. PMC2951171. PMID 20929891.
  103. ^ Nassisi D, Oishi ML (January 2012). "Evidence-based guidelines for evaluation and antimicrobial therapy for common emergency department infections". Emergency Medicine Exercise. fourteen (1): 1–28, quiz 28–29. PMID 22292348.
  104. ^ a b Sajadi MM, Bonabi R, Sajadi MR, Mackowiak PA (October 2012). "Akhawayni and the showtime fever curve". Clinical Infectious Diseases. 55 (7): 976–80. doi:10.1093/cid/cis596. PMID 22820543.
  105. ^ Casanova, Jean-Laurent; Abel, Laurent (2021). "Lethal Infectious Diseases as Inborn Errors of Immunity: Toward a Synthesis of the Germ and Genetic Theories". Annual Review of Pathology: Mechanisms of Disease. xvi: 23–fifty. doi:10.1146/annurev-pathol-031920-101429. PMC7923385. PMID 32289233.
  106. ^ Straus, Bernard (September 1970) [start presented at a conference on 27 May 1970]. "Defunct and Dying Diseases" (PDF). Bulletin of the New York University of Medicine. 46 (ix): 686–706. PMC1749762. PMID 4916301. Archived from the original on 6 February 2020.
  107. ^ a b c d Crocetti Yard, Moghbeli N, Serwint J (June 2001). "Fever Phobia Revisited: Have Parental Misconceptions About Fever Changed in 20 Years?". Pediatrics. 107 (6): 1241–1246. doi:10.1542/peds.107.6.1241. PMID 11389237. Retrieved 31 March 2020.
  108. ^ Klass, Perri (10 January 2011). "Lifting a Veil of Fear to See a Few Benefits of Fever". The New York Times. Archived from the original on 29 September 2015.
  109. ^ "Equusite Vital Signs". equusite.com. Archived from the original on 26 March 2010. Retrieved 22 March 2010.
  110. ^ Schmidt-Nielsen K, Schmidt-Nielsen B, Jarnum SA, Houpt TR (January 1957). "Body temperature of the camel and its relation to water economic system". The American Journal of Physiology. 188 (1): 103–12. doi:10.1152/ajplegacy.1956.188.1.103. PMID 13402948.
  111. ^ Leese A (March 1917). ""Tips" on camels, for veterinarian surgeons on agile service". The British Veterinarian Journal. 73: 81 – via Google Books.
  112. ^ Tefera Yard (July 2004). "Observations on the clinical exam of the camel (Camelus dromedarius) in the field". Tropical Animal Health and Product. 36 (5): 435–49. doi:10.1023/b:trop.0000035006.37928.cf. PMID 15449833. S2CID 26358556.
  113. ^ a b Thomas MB, Blanford S (July 2003). "Thermal biological science in insect-parasite interactions". Trends in Ecology & Evolution. 18 (7): 344–fifty. doi:10.1016/S0169-5347(03)00069-7.

Farther reading [edit]

  • Rhoades R, Pflanzer RG (1996). "Chapter 27: Regulation of Body Temperature (Clinical Focus: Pathogenesis of Fever)". Human Physiology (3rd ed.). Philadelphia, PA: Saunders College. ISBN9780030051593 . Retrieved 2 April 2020. {{cite volume}}: CS1 maint: uses authors parameter (link)

External links [edit]

  • Fever and Taking Your Child's Temperature
  • US National Establish of Wellness factsheet
  • Drugs nigh commonly associated with the adverse outcome Pyrexia (Fever) as reported the FDA
  • Fever at MedlinePlus
  • Why are We Then Agape of Fevers? at The New York Times

langderpromed.blogspot.com

Source: https://en.wikipedia.org/wiki/Fever

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